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Gillian Bates
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Gillian Bates

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Gillian Bates
Born (1956-05-19) 19 May 1956
Nationality British
Alma mater
Known for Co-discovering cause of Huntington's disease
Awards
Scientific career
Fields Neurogenetics
Institutions
Thesis Molecular approaches towards the fine genetic mapping of human chromosomes : with special reference to the cystic fibrosis gene (1987)
Website www.ucl.ac.uk/ion/departments/sobell/Research/GBates

Gillian Patricia Bates (born 19 May 1956)FMedSci FRS is a British biologist. She is distinguished for her research into the molecular basis of Huntington's disease and in 1998 was awarded the GlaxoSmithKline Prize as a co-discoverer of the cause of this disease. As of 2016, she is Professor of Neurogenetics at UCL Institute of Neurology and the co-director of UCL Huntington's Disease Centre.

Education

Bates was educated at Kenilworth Grammar School and the University of Sheffield where she graduated with a Bachelor of Science degree in 1979. She completed her postgraduate study at Birkbeck College, London where she was awarded a Master of Science degree in 1984 followed by St Mary's Hospital Medical School where she was awarded a PhD in 1987 for genetic mapping of the cystic fibrosis gene, working in the lab of Robert Williamson.

Research

Bates's research has focused on Huntington's disease. She was one of the group who first cloned the Huntington's disease gene. She also created the first mouse model of the disease, the R6/2 mouse, an important step in understanding the pathogenesis of Huntington's.

Prior to joining UCL in 2016, Bates was the head of the Neurogenetics Research Group at King's College London.

Awards and honours

Bates has been elected a fellow of the Academy of Medical Sciences (1999) and a member of the European Molecular Biology Organisation (2002). She was elected a Fellow of the Royal Society in 2007 and to its Council in 2011. In 1998, she was awarded the Royal Society Glaxo Wellcome Award jointly with Stephen Davies, for the "discovery of the cause of Huntington's Disease".


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